CHO to avoid sustained ketosis. • Typical intake = 15–20% energy from CHO (<2.5 g −1 ·kg −1 ·day −1 ), 15–20% protein, 60–65% fat in combination with a moderate-endurance training volume (>5 hr/week). • Deprivation of CHO for muscle fuel needs while consuming high amounts of dietary fat causes
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Louise M. Burke, John A. Hawley, Asker Jeukendrup, James P. Morton, Trent Stellingwerff, and Ronald J. Maughan
Joanne G. Mirtschin, Sara F. Forbes, Louise E. Cato, Ida A. Heikura, Nicki Strobel, Rebecca Hall, and Louise M. Burke
moderate protein intake (<∼2 g/kg BM) to achieve chronic ketosis requires the elimination of many staple foods from the standard Western diet (e.g., most fruit, starchy vegetables and legumes, cereal products) and restrictions on the serving sizes of others. For example, there are portion limits on meats
Andrzej Gawrecki, Aleksandra Araszkiewicz, Agnieszka Szadkowska, Grzegorz Biegański, Jan Konarski, Katarzyna Domaszewska, Arkadiusz Michalak, Bogda Skowrońska, Anna Adamska, Dariusz Naskręt, Przemysława Jarosz-Chobot, Agnieszka Szypowska, Tomasz Klupa, and Dorota Zozulińska-Ziółkiewicz
activity among diabetic patients, it was very important to develop safety rules for players during a mass sports event. The most important goal for medical care was to avoid decompensation of diabetes, that is, severe hypoglycemia or hyperglycemia with ketosis. A greater risk of ketoacidosis occurs in
Trent Stellingwerff, James P. Morton, and Louise M. Burke
% energy from CHO to prevent ketosis while reducing CHO intake to levels below the fuel costs of daily training) and the more restrictive ketogenic LCHF diet (K-LCHF: typically <50 g/day CHO and 75–80% fat; Burke et al., 2018 ). However, studies have shown that as little as 5 days of exposure to HFLC
Romain Meeusen and Lieselot Decroix
nutritional ketosis on cognitive outcomes in mild to moderate AD and in mild cognitive impairment. While this effect may be attributable in part to correction of hyperinsulinemia, other mechanisms associated with ketosis, such as reduced inflammation and enhanced energy metabolism, also may have contributed
(0.4[0.2–1.0]) ( P = .008) and between PR2 (0.4[0.2–1.0]) and the marathon (0.1[0–0.3]) ( P = .008). There was no severe hypoglycemia or hyperglycemia with ketosis at any time before, during and after the PRs and Marathon. Conclusions.— To avoid exercise-induced hypoglycemia, it is necessary to reduce usual doses
Katherine Elizabeth Black, Alistair David Black, and Dane Frances Baker
.J. , Gervino , E. , & Blackburn , G.L. ( 1983 ). The human metabolic response to chronic ketosis without caloric restriction: Preservation of submaximal exercise capability with reduced carbohydrate oxidation . Metabolism 32 ( 8 ), 769 – 776 . PubMed ID: 6865776 doi:10
Claire E. Badenhorst, Katherine E. Black, and Wendy J. O’Brien
metabolism. During periods of starvation or LEA, the increase in PPARGC1α will activate gluconeogenesis, ketosis, and β-oxidation of fatty acids at the liver ( Lin, Handschin, & Spiegelman, 2005 ; Scarpulla, 2008 ). Interestingly, during prolonged starvation (48 hr) in mice, no detectable increases in IL-6
Gary J. Slater, Jennifer Sygo, and Majke Jorgensen
only following severe dietary carbohydrate restriction, sufficient to promote a state of ketosis. Such a state is unlikely among competitive athletes tapering prior to competition who follow a meal plan with even a moderate carbohydrate content. In light of this, sprint athletes are advised to choose a
), incorporating MCT into the diet may allow more carbohydrates yet preserving ketosis and exert less inhibitory effect on muscle glucose metabolism. The purpose of this study was therefore to examine the effects of long-term feeding of ketogenic diet containing MCT on key metabolic enzymes in rat skeletal muscle
