% energy from CHO to prevent ketosis while reducing CHO intake to levels below the fuel costs of daily training) and the more restrictive ketogenic LCHF diet (K-LCHF: typically <50 g/day CHO and 75–80% fat; Burke et al., 2018 ). However, studies have shown that as little as 5 days of exposure to HFLC
Search Results
Trent Stellingwerff, James P. Morton, and Louise M. Burke
(0.4[0.2–1.0]) ( P = .008) and between PR2 (0.4[0.2–1.0]) and the marathon (0.1[0–0.3]) ( P = .008). There was no severe hypoglycemia or hyperglycemia with ketosis at any time before, during and after the PRs and Marathon. Conclusions.— To avoid exercise-induced hypoglycemia, it is necessary to reduce usual doses
Gary J. Slater, Jennifer Sygo, and Majke Jorgensen
only following severe dietary carbohydrate restriction, sufficient to promote a state of ketosis. Such a state is unlikely among competitive athletes tapering prior to competition who follow a meal plan with even a moderate carbohydrate content. In light of this, sprint athletes are advised to choose a
Claire E. Badenhorst, Katherine E. Black, and Wendy J. O’Brien
metabolism. During periods of starvation or LEA, the increase in PPARGC1α will activate gluconeogenesis, ketosis, and β-oxidation of fatty acids at the liver ( Lin, Handschin, & Spiegelman, 2005 ; Scarpulla, 2008 ). Interestingly, during prolonged starvation (48 hr) in mice, no detectable increases in IL-6
), incorporating MCT into the diet may allow more carbohydrates yet preserving ketosis and exert less inhibitory effect on muscle glucose metabolism. The purpose of this study was therefore to examine the effects of long-term feeding of ketogenic diet containing MCT on key metabolic enzymes in rat skeletal muscle
Oliver J. Peacock, Javier T. Gonzalez, Simon P. Roberts, Alan Smith, Scott Drawer, and Keith A. Stokes
and both made an estimated 16 kJ/kg body mass (BM) available for metabolism (total energy intake 1,528 ± 145 kJ). The ketone monoester was provided at a total dose of 590 mg/kg BM based on pilot data showing that this dosing level induces a sustained moderate ketosis (blood BHB of ∼2–3 mmol/L) that
Devin G. McCarthy, Jack Bone, Matthew Fong, Phillippe J.M. Pinckaers, William Bostad, Douglas L. Richards, Luc J.C. van Loon, and Martin J. Gibala
Nutritional ketosis induced through the ingestion of ketogenic supplements can alter physiological responses to exercise ( Evans et al., 2017 ). This practice has also been purported to enhance performance, at least under selected conditions, although the precise mechanistic basis is unclear
Alannah K.A. McKay, Megan L.R. Ross, Nicolin Tee, Avish P. Sharma, Jill J. Leckey, and Louise M. Burke
perceived exertion per kilometer travelled and a higher perceptual (RPE) and physiological (HR) effort for any given speed. The LCHF diet was associated with a sustained increase in concentrations of blood β HB concentrations above the level considered to signal ketosis (0.5 mM) 2 and within proposed
Louise M. Burke, John A. Hawley, Asker Jeukendrup, James P. Morton, Trent Stellingwerff, and Ronald J. Maughan
CHO to avoid sustained ketosis. • Typical intake = 15–20% energy from CHO (<2.5 g −1 ·kg −1 ·day −1 ), 15–20% protein, 60–65% fat in combination with a moderate-endurance training volume (>5 hr/week). • Deprivation of CHO for muscle fuel needs while consuming high amounts of dietary fat causes
Joanne G. Mirtschin, Sara F. Forbes, Louise E. Cato, Ida A. Heikura, Nicki Strobel, Rebecca Hall, and Louise M. Burke
moderate protein intake (<∼2 g/kg BM) to achieve chronic ketosis requires the elimination of many staple foods from the standard Western diet (e.g., most fruit, starchy vegetables and legumes, cereal products) and restrictions on the serving sizes of others. For example, there are portion limits on meats
