Team captains play an important role in promoting positive life-skills development (PLSD) in their teammates. However, little research has been conducted to understand how team captains perceive the value of PLSD in high-performance sport. Therefore, the purpose of the present study was to understand how team captains integrate PLSD in high-performance sport. The participants in this study were 10 team captains from high-performance sports with teammates ranging from 14 to 38 years old. Data collection was conducted through 2 semistructured interviews. Results indicated that participants considered themselves PLSD-focused leaders and acknowledged the need to develop specific PLSD strategies. Nevertheless, team captains recognized the need to obtain support from their coaches to implement PLSD. Moving forward, coaches could provide a support system for athlete leaders to further enhance their ability to promote PLSD in high-performance sport.
Fernando Santos, Leisha Strachan, Daniel Gould, Paulo Pereira and Cláudia Machado
Maximiliano I. Schaun, Leonardo Lisboa Motta, Rayane Teixeira, Fábio Klamt, Juliane Rossato, Alexandre Machado Lehnen, Maria Cláudia Irigoyen and Melissa M. Markoski
In acute myocardial infarction (AMI), reactive oxygen species may cause irreversible damage to the heart tissue. Physical training is capable of enhancing antioxidant capacity, acting as a cardioprotective factor. We assessed the preventive effects of physical training on the antioxidant and functional responses of the heart of Wistar Kyoto rats after AMI. Wistar Kyoto rats (n = 12) were allocated to sedentary (SED) or trained (EXE—aerobic training on a treadmill) groups. Echocardiographic exams were performed 48 hr before and 48 hr after the induction of AMI. Superoxide dismutase (SOD) and catalase (CAT) activities, and total glutathione (GSH) were measured in vitro in the heart tissue. After AMI, the EXE group showed higher left ventricular shortening fraction (29%; p = .004), higher cardiac output (37%; p = .032) and reduced myocardial infarction size (16%; p = .007) than SED. The EXE group showed a higher nonenzymatic antioxidant capacity (GSH, 23%; p = .004), but the SOD and CAT activities were higher in SED (23% SOD; p = .021 and 20% CAT; p = .016). In addition, the SOD activity was positively correlated with myocardial infarction size and inversely correlated with cardiac output. Physical training partially preserved cardiac function and increased intracellular antioxidant response in cardiac tissue of animals after AMI.