Search Results

Purpose: To compare the effects of graded hypoxia during exhaustive intermittent cycling on subsequent rapid and maximal torque-production capacity. Methods: Fifteen well-trained cyclists repeated intermittent cycling bouts (15 s at 30% of anaerobic power reserve; rest = 45 s) until exhaustion at sea level (FiO₂ ∼0.21/end-exercise arterial oxygen saturation ∼96%), moderate hypoxia (FiO₂ ∼0.16/∼90%), and severe hypoxia (FiO₂ ∼0.12/∼79%). Rapid (rate of torque development [RTD]) and maximal isometric torque-production capacities of the knee extensors were assessed at baseline (visit 1) and exhaustion (visits 2–4). Results: Exercise capacity decreased with hypoxia severity (39 [30], 22 [13], and 13 [6] cycle efforts in sea level, moderate hypoxia, and severe hypoxia, respectively; P = .002). Changes in maximal-voluntary-contraction torque between baseline and postexercise in all conditions were not statistically significant (pooled values: −2.6% [5.7%]; P = .162). Peak RTD measured postexercise was reduced below baseline in all conditions (–21.5% [5.1%]; P ≤ .015). Compared with baseline, absolute RTD values were lower at 0- to 30-millisecond (–35.1% [5.3%], P ≤ .020), 0- to 50-millisecond (–40.0% [3.9%], P ≤ .002), 0- to 100-millisecond (–30.7% [3.7%], P ≤ .001), and 0- to 200-millisecond (–18.1% [2.4%], P ≤ .004) time intervals in all conditions. Conclusions: Exhaustive intermittent cycling induces substantial yet comparable impairments in RTD of knee extensors between normoxia and moderate to severe hypoxia.

Purpose: To quantify the demands of specific on- and off-court sessions, using internal and external training load metrics, in elite squash. Methods: A total of 15 professional squash players (11 males and 4 females) wore a 100-Hz triaxial accelerometer/global positioning system unit and heart rate monitor during on-court “Group,” “Feeding,” “Ghosting,” “Matchplay,” and off-court “Conditioning” sessions across a 2-week in-season microcycle. Comparisons of absolute training load (total values) and relative intensity (per minute) were made between sessions for internal (session rating of perceived exertion, differential rating of perceived exertion, TRIMP) and external (Playerload, very high–intensity movements [>3.5 m·s⁻²]) metrics. Results: The Group sessions were the longest (79 [12] min), followed by Feeding (55 [15] min), Matchplay (46 [17] min), Conditioning (37 [9] min), and Ghosting (35 [6] min). Time >90% maximum heart rate was the lowest during Feeding (vs all others P < .05) but other sessions were not different (all P > .05). Relative Playerload during Conditioning (14.3 [3.3] arbitrary unit [a.u.] per min, all P < .05) was higher than Ghosting (7.5 [1.2] a.u./min) and Matchplay (6.9 [1.5] a.u./min), with no difference between these 2 sessions (P ≥ .999). Conditioning produced the highest Playerloads (519 [153] a.u., all P < .001), with the highest on-court Playerloads from Group (450 [94] a.u., all P < .001). The highest session rating of perceived exertion (all P < .001), Edward’s TRIMP (all P < .001), and TEAM-TRIMP (all P < .019) occurred during the Group sessions. Conclusions: Squash Matchplay does not systematically produce the highest training intensities and loads. Group sessions provide the highest training loads for many internal and external parameters and, therefore, play a central role within the training process. These findings facilitate planning or adjustment of intensity, volume, and frequency of sessions to achieve desirable physical outcomes.

Background : Compared with normoxia, repeated short (5–10 s) sprints (>10 efforts) with incomplete recovery (≤30 s) in hypoxia likely cause substantial performance reduction accompanied by larger metabolic disturbances and magnitude of neuromuscular fatigue. However, the effects of hypoxia on performance of repeated long (30 s) “all-out” efforts with near complete recovery (4.5 min) and resulting metabolic and neuromuscular adjustments remain unclear. Purpose : The intention was to compare acute performance, metabolic, and neuromuscular responses across repeated Wingates between hypoxia and normoxia. Methods : On separate visits, 6 male participants performed 4 × 30-second Wingate efforts with 4.5-minute recovery in either hypoxia (fraction of inspired oxygen: 0.145) or normoxia. Responses to exercise (muscle and arterial oxygenation trends, heart rate, and blood lactate concentration) and the integrity of neuromuscular function in the knee extensors were assessed for each exercise bout. Results : Mean (P = .80) and peak (P = .92) power outputs, muscle oxygenation (P = .88), blood lactate concentration (P = .72), and perceptual responses (all Ps > .05) were not different between conditions. Arterial oxygen saturation was significantly lower, and heart rate higher, in hypoxia versus normoxia (P < .001). Maximal voluntary contraction force and peripheral fatigue indices (peak twitch force and doublets at low and high frequencies) decreased across efforts (all Ps < .001) irrespective of conditions (all Ps > .05). Conclusion : Despite heightened arterial hypoxemia and cardiovascular solicitation, hypoxic exposure during 4 repeated 30-second Wingate efforts had no effect on performance and accompanying metabolic and neuromuscular adjustments.

Background: Training in hypoxia versus normoxia often induces larger physiological adaptations, while this does not always translate into additional performance benefits. A possible explanation is a reduced oxygen flux, negatively affecting training intensity and/or volume (decreasing training stimulus). Repeated Wingates (RW) in normoxia is an efficient training strategy for improving both physiological parameters and exercise capacity. However, it remains unclear whether the addition of hypoxia has a detrimental effect on RW performance. Purpose: To test the hypothesis that acute moderate hypoxia exposure has no detrimental effect on RW, while both metabolic and perceptual responses would be slightly higher. Methods: On separate days, 7 male university sprinters performed 3 × 30-s Wingate efforts with 4.5-min passive recovery in either hypoxia (FiO₂: 0.145) or normoxia (FiO₂: 0.209). Arterial oxygen saturation was assessed before the first Wingate effort, while blood lactate concentration and ratings of perceived exertion were measured after each bout. Results: Mean (P = .92) and peak (P = .63) power outputs, total work (P = .98), and the percentage decrement score (P = .25) were similar between conditions. Arterial oxygen saturation was significantly lower in hypoxia versus normoxia (92.0% [2.8%] vs 98.1% [0.4%], P < .01), whereas blood lactate concentration (P = .78) and ratings of perceived exertion (P = .51) did not differ between conditions. Conclusion: In sprinters, acute exposure to moderate hypoxia had no detrimental effect on RW performance and associated metabolic and perceptual responses.

Purpose: To quantify the acute effects of a spectrum of eccentric load reductions on neuromechanical adjustments during the performance of weighted jump squats (WJSs). Methods: On separate days, 16 well-trained participants performed WJS trials with various eccentric load reductions (0% [body mass only], 25%, 50%, 75%, and 100% [standard WJS] of concentric load) with a mechanical braking unit, while concentric load was set at 30% of peak isometric squat force in all trials. A force platform and a motion-capture system were used to assess neuromuscular performance. Results: Peak power output was 6.2% (4.7%) higher when load was reduced by 50% versus 0% (55.4 [7.8] vs 51.9 [7.6] W/kg; P = .001). Compared with no braking (0.326 [0.059] m), jump height was ∼13% to 17% higher for all eccentric load reduction conditions (all P < .001). Vertical ground reaction forces were progressively lower for 25%, 50%, 75%, and 100% loading conditions (–22.1% [14.6%], –32.3% [10.8%], –42.0% [13.2%], and –46.1% [14.7%]; all P ≤ .001) in reference to body mass only. Conclusion: Eccentric load reduction is advantageous compared with traditional isoinertial loading for improving both jump height and peak power output during the concentric portion of maximal-effort WJS. This practice also decreases mechanical constraints in the lower extremities, which may become beneficial for load-compromised individuals.

Purpose: To quantify match load associated with padel and compare responses with both singles and doubles tennis. Methods: On separate days, 12 participants (7 men and 5 women) played 60-minute padel (PADEL), singles tennis (SINGLES), and doubles tennis (DOUBLES) simulated games. Participants wore a 10-Hz GPS/100-Hz triaxial accelerometer unit and heart-rate monitor. Exercise-related sensations and blood lactate concentration were monitored every 20 minutes. Match-play characteristics (temporal structure) and shot selection were derived from video analysis. Vertical jump ability was assessed before and after each game. Results: Heart rate, exercise-related sensations (overall perceived exertion and limb discomfort), and physical load (total distance covered, PlayerLoad, acceleration density and load) for SINGLES were higher compared with DOUBLES and PADEL (all P ≤ .05). Blood lactate concentrations remained low (1–2 mmol·L⁻¹) and did not differ between conditions. Effective playing time (P < .001) was lower in SINGLES and DOUBLES compared with PADEL. The number of forehands (P = .002) and backhands (P < .001) was greater for SINGLES than for DOUBLES and PADEL. The number of volleys/smashes and lobs (P < .05) was greater for PADEL compared with SINGLES and DOUBLES. Performance for squat, countermovement, and multirebound jumps was similarly reduced below baseline after match play (P < .05), independent of condition. Conclusion: Padel imposes a unique match load on players that is different from singles tennis and more closely resembles that of doubles tennis. Cardiovascular stimulation and physical load are highest in singles tennis, while padel sees players hit a larger variety of shots with higher effective playing percentages.

Purpose : The authors compared the effects of active preconditioning with local and systemic hypoxia during submaximal cycling. Methods : On separate visits, 14 active participants completed 4 trials. Each visit was composed of 1 preconditioning phase followed, after 40 minutes of rest, by 3 × 6-minute cycling bouts (intensity = 85% of critical power; rest = 6 min). The preconditioning phase consisted of 4 × 5-minute cycling bouts at 1.5 W·kg⁻¹ (rest = 5 min) in 4 conditions: control (no occlusion and normoxia), blood flow restriction (60% of total occlusion), HYP (systemic hypoxia; inspired fraction of oxygen = 13.6%), and blood flow restriction + HYP (local and systemic hypoxia combined). Results : During the preconditioning phase, there were main effects of both systemic (all P < .014) and local hypoxia (all P ≤ .001) on heart rate, arterial oxygen saturation, leg discomfort, difficulty of breathing, and blood lactate concentration. Cardiorespiratory variables, gross efficiency, energy cost, and energy expenditure during the last minute of 6-minute cycling bouts did not differ between conditions (all P > .105). Conclusion : Local and systemic hypoxic stimuli, or a combination of both, during active preconditioning did not improve physiological responses such as cycling efficiency during subsequent submaximal cycling.