The aims of the study were to investigate the differences in kinematics and kinetics between the dominant and nondominant leg during single-leg jumps without arm swing, and to determine the relationship between bilateral asymmetry in isokinetic knee strength and the single-leg jump. Isokinetic knee strength and single-leg jump kinematics and kinetics were measured in 11 male participants. The bilateral asymmetry index was calculated for each parameter. For isokinetic knee strength, there were no significant differences between the dominant and nondominant legs. Significant correlations were observed for the bilateral asymmetry index for isokinetic knee strength at 180 degrees per second and the bilateral asymmetry indexes for maximum flexion angle and the mean knee joint torque during the single-leg jumps. In conclusion, the findings of the current study suggest an association between knee strength imbalances and the joint angle, as well as the torque produced in single-leg jumps, although no relationship between knee strength and jump height was observed.
Yuji Kobayashi, Junjiro Kubo, Takeo Matsubayashi, Akifumi Matsuo, Kando Kobayashi and Naokata Ishii
Yoshiharu Shimomura, Asami Inaguma, Satoko Watanabe, Yuko Yamamoto, Yuji Muramatsu, Gustavo Bajotto, Juichi Sato, Noriko Shimomura, Hisamine Kobayashi and Kazunori Mawatari
The authors examined the effect of branched-chain amino acid (BCAA) supplementation on squat-exercise-induced delayed-onset muscle soreness (DOMS) using 12 young, healthy, untrained female participants. The experiment was conducted with a crossover double-blind design. In the morning on the exercise-session day, the participants ingested either BCAA (isoleucine:leucine:valine = 1:2.3:1.2) or dextrin at 100 mg/kg body weight before the squat exercise, which consisted of 7 sets of 20 squats/set with 3-min intervals between sets. DOMS showed a peak on Days 2 and 3 in both trials, but the level of soreness was significantly lower in the BCAA trial than in the placebo. Leg-muscle force during maximal voluntary isometric contractions was measured 2 d after exercise (Day 3), and the BCAA supplementation suppressed the muscle-force decrease (to ~80% of the value recorded under the control conditions) observed in the placebo trial. Plasma BCAA concentrations, which decreased after exercise in the placebo trial, were markedly elevated during the 2 hr postexercise in the BCAA trial. Serum myoglobin concentration was increased by exercise in the placebo but not in the BCAA trial. The concentration of plasma elastase as an index of neutrophil activation appeared to increase after the squat exercise in both trials, but the change in the elastase level was significant only in the placebo trial. These results suggest that muscle damage may be suppressed by BCAA supplementation.