overuse injuries, efficient muscle recovery is of special importance during such periods. Although the cellular mechanisms driving the acute regenerative processes are not well elucidated, a growing number of studies have unveiled the benefits of protein feeding strategies in regard to optimizing recovery
Mads S. Larsen, Dagmar Clausen, Astrid Ank Jørgensen, Ulla R. Mikkelsen and Mette Hansen
Joanne L. Fallowfield and Clyde Williams
The influence of increased carbohydrate intake on endurance capacity was investigated following a bout of prolonged exercise and 22.5 hrs of recovery. Sixteen male subjects were divided into two matched groups, which were then randomly assigned to either a control (C) or a carbohydrate (CHO) condition. Both groups ran at 70% VO2max on a level treadmill for 90 min or until volitional fatigue, whichever came first (T1), and 22.5 hours later they ran at the same % VO2max for as long as possible to assess endurance capacity (T2). During the recovery, the carbohydrate intake of the CHO group was increased from 5.8 (±0.5) to 8.8 (±0.1) g kg-1 BW. This was achieved by supplementing their normal diet with a 16.5% glucose-polymer solution. An isocaloric diet was prescribed for the C group, in which additional energy was provided in the form of fat and protein. Run times over T1 did not differ between the groups. However, over T2 the run time of the C group was reduced by 15.57 min (p<0.05), whereas those in the CHO group were able to match their T1 performance. Blood glucose remained stable throughout Tl and T2 in both groups. In contrast, blood lactate, plasma FFA, glycerol, ammonia, and urea increased. Thus, a high carbohydrate diet restored endurance capacity within 22.5 hrs whereas an isocaloric diet without additional carbohydrate did not.
Dean Norris, David Joyce, Jason Siegler, James Clock and Ric Lovell
constraints, longitudinal measurement of many of these markers is rarely feasible, as is finding a singular metric that is indicative of all fatigue domains. Of these markers, however, recovery of NF is accepted as one of the most practically viable due to its relative ease of assessment and reported
Psychological skills such as goal setting, imagery, relaxation and self-talk have been used in performance enhancement, emotional regulation, and increasing one’s confidence and/or motivation in sport. These skills can also be applied with athletes during recovery from injury in the rehabilitation setting or in preseason meetings for preventing injury. Research on psychological skill use with athletes has shown that such skills have helped reduce negative psychological outcomes, improve coping skills, and reduce reinjury anxiety (Evans & Hardy, 2002; Johnson, 2000; Mankad & Gordon, 2010). Although research has been limited in psychological skill implementation with injured athletes, these skills can be used when working with injured athletes or in the prevention of injury. Injured athletes may use psychological skills such as setting realistic goals in coming back from injury, imagery to facilitate rehabilitation, and relaxation techniques to deal with pain management. In prevention of injury, the focus is on factors that put an individual at-risk for injury. Thus, teaching strategies of goal setting, imagery, relaxation techniques, and attention/focus can be instrumental in preparing athletes for a healthy season.
Ian Rollo, Franco M. Impellizzeri, Matteo Zago and F. Marcello Iaia
The physical-performance profiles of subelite male footballers were monitored during 6 wk of a competitive season. The same squad of players played either 1 (1G, n = 15) or 2 (2G, n = 15) competitive matches per week. On weeks 0, 3, and 6, 48 h postmatch, players completed countermovement jump (CMJ), 10- and 20-m sprints, the Yo-Yo Intermittent Recovery Test (YYIRT), and the Recovery-Stress Questionnaire. Both groups undertook 2 weekly training sessions. The 2G showed after 6 wk lower YYIRT (–11% to 3%, 90% CI –15.8% to –6.8%; P < .001) and CMJ performances (–18.7%, –21.6 to –15.9%; P = .007) and higher 10-m (4.4%, 1.8–6.9%; P = .007) and 20-m sprints values (4.7%, 2.9% to 6.4%; P < .001). No differences were found at 3 wk (.06 < P < .99). No changes over time (.169 < P < .611) and no differences time × group interactions (.370 < P < .550) were found for stress, recovery, and the Stress Recovery Index. In conclusion players’ ability to sprint, jump, and perform repeated intense exercise was impaired when playing 2 competitive matches a week over 6 wk.
David N. Borg, Ian B. Stewart, John O. Osborne, Christopher Drovandi, Joseph T. Costello, Jamie Stanley and Geoffrey M. Minett
circumvent errors in exercise prescription. This is of importance, as errors in prescription that result in an imbalance between training and recovery could lead to nonfunctional overreaching and diminish performance gains. 4 – 6 Traditional heat-based training methods have utilized exercise in a hot
Nuttaset Manimmanakorn, Jenny J. Ross, Apiwan Manimmanakorn, Samuel J.E. Lucas and Michael J. Hamlin
To compare whole-body vibration (WBV) with traditional recovery protocols after a high-intensity training bout.
In a randomized crossover study, 16 athletes performed 6 × 30-s Wingate sprints before completing either an active recovery (10 min of cycling and stretching) or WBV for 10 min in a series of exercises on a vibration platform. Muscle hemodynamics (assessed via near-infrared spectroscopy) were measured before and during exercise and into the 10-min recovery period. Blood lactate concentration, vertical jump, quadriceps strength, flexibility, rating of perceived exertion (RPE), muscle soreness, and performance during a single 30-s Wingate test were assessed at baseline and 30 and 60 min postexercise. A subset of participants (n = 6) completed a 3rd identical trial (1 wk later) using a passive 10-min recovery period (sitting).
There were no clear effects between the recovery protocols for blood lactate concentration, quadriceps strength, jump height, flexibility, RPE, muscle soreness, or single Wingate performance across all measured recovery time points. However, the WBV recovery protocol substantially increased the tissue-oxygenation index compared with the active (11.2% ± 2.4% [mean ± 95% CI], effect size [ES] = 3.1, and –7.3% ± 4.1%, ES = –2.1 for the 10 min postexercise and postrecovery, respectively) and passive recovery conditions (4.1% ± 2.2%, ES = 1.3, 10 min postexercise only).
Although WBV during recovery increased muscle oxygenation, it had little effect in improving subsequent performance compared with a normal active recovery.
Daniel Viggiani and Jack P. Callaghan
hip extensor musculature in clinical LBP populations arises from the interplay between hip function and LBP. 24 , 25 While LBP can also slow the recovery from muscle fatigue of lumbar spine extensor musculature, 26 – 28 it is not known how LBP affects recovery from hip muscle fatigue. LBP
Repeated-sprint ability (RSA) is now well accepted as an important fitness component in team-sport performance. It is broadly described as the ability to perform repeated short (~3–4 s, 20–30 m) sprints with only brief (~10–30 s) recovery between bouts. Over the past 25 y a plethora of RSA tests have been trialed and reported in the literature. These range from a single set of ~6–10 short sprints, departing every 20–30 s, to team-sport game simulations involving repeating cycles of walk-jog-stride-sprint movements over 45–90 min. Such a wide range of RSA tests has not assisted the synthesis of research findings in this area, and questions remain regarding the optimal methods of training to best improve RSA. In addition, how RSA test scores relate to player “work rate,” match performance, or both requires further investigation to improve the application of RSA testing and training to elite team-sport athletes.
Jamie Douglas, Daniel J. Plews, Phil J. Handcock and Nancy J. Rehrer
To determine whether a facilitated recovery via cold-water immersion (CWI) after simulated rugby sevens would influence parasympathetic reactivation and repeated-sprint (RS) performance across 6 matches in a 2-d tournament.
Ten male team-sport athletes completed 6 rugby sevens match simulations over 2 d with either postmatch passive recovery (PAS) or CWI in a randomized crossover design. Parasympathetic reactivation was determined via the natural logarithm of the square root of the mean of the sum of the squares of differences between adjacent R-R intervals (ln rMSSD). RS performance was calculated as time taken (s) to complete 6 × 30-m sprints within the first half of each match.
There were large increases in postintervention ln rMSSD between CWI and PAS after all matches (ES 90% CL: +1.13; ±0.21). Average heart rate (HR) during the RS performance task (HRAverage RS) was impaired from baseline from match 3 onward for both conditions. However, HRAverage RS was higher with CWI than with PAS (ES 90% CL: 0.58; ±0.58). Peak HR during the RS performance task (HRPeak RS) was similarly impaired from baseline for match 3 onward during PAS and for match 4 onward with CWI. HRPeak RS was very likely higher with CWI than with PAS (ES 90% CL: +0.80; ±0.56). No effects of match or condition were observed for RS performance, although there were moderate correlations between the changes in HRAverage RS (r 90% CL: –0.33; ±0.14), HRPeak RS (r 90% CL: –0.38; ±0.13), and RS performance.
CWI facilitated cardiac parasympathetic reactivation after a simulated rugby sevens match. The decline in average and peak HR across matches was partially attenuated by CWI. This decline was moderately correlated with a reduction in RS performance.