immunity, potentially leaving the individual open to opportunistic pathogenic agents and suboptimal recovery processes ( Russo et al., 2019 ; Walsh, 2018 ). Furthermore, neutrophil chemotaxis, phagocytic ability, pathogenic termination, and/or tissue debris demolition functions (e
Ricardo J.S. Costa, Vera Camões-Costa, Rhiannon M.J. Snipe, David Dixon, Isabella Russo and Zoya Huschtscha
Psychological skills such as goal setting, imagery, relaxation and self-talk have been used in performance enhancement, emotional regulation, and increasing one’s confidence and/or motivation in sport. These skills can also be applied with athletes during recovery from injury in the rehabilitation setting or in preseason meetings for preventing injury. Research on psychological skill use with athletes has shown that such skills have helped reduce negative psychological outcomes, improve coping skills, and reduce reinjury anxiety (Evans & Hardy, 2002; Johnson, 2000; Mankad & Gordon, 2010). Although research has been limited in psychological skill implementation with injured athletes, these skills can be used when working with injured athletes or in the prevention of injury. Injured athletes may use psychological skills such as setting realistic goals in coming back from injury, imagery to facilitate rehabilitation, and relaxation techniques to deal with pain management. In prevention of injury, the focus is on factors that put an individual at-risk for injury. Thus, teaching strategies of goal setting, imagery, relaxation techniques, and attention/focus can be instrumental in preparing athletes for a healthy season.
Nuttaset Manimmanakorn, Jenny J. Ross, Apiwan Manimmanakorn, Samuel J.E. Lucas and Michael J. Hamlin
To compare whole-body vibration (WBV) with traditional recovery protocols after a high-intensity training bout.
In a randomized crossover study, 16 athletes performed 6 × 30-s Wingate sprints before completing either an active recovery (10 min of cycling and stretching) or WBV for 10 min in a series of exercises on a vibration platform. Muscle hemodynamics (assessed via near-infrared spectroscopy) were measured before and during exercise and into the 10-min recovery period. Blood lactate concentration, vertical jump, quadriceps strength, flexibility, rating of perceived exertion (RPE), muscle soreness, and performance during a single 30-s Wingate test were assessed at baseline and 30 and 60 min postexercise. A subset of participants (n = 6) completed a 3rd identical trial (1 wk later) using a passive 10-min recovery period (sitting).
There were no clear effects between the recovery protocols for blood lactate concentration, quadriceps strength, jump height, flexibility, RPE, muscle soreness, or single Wingate performance across all measured recovery time points. However, the WBV recovery protocol substantially increased the tissue-oxygenation index compared with the active (11.2% ± 2.4% [mean ± 95% CI], effect size [ES] = 3.1, and –7.3% ± 4.1%, ES = –2.1 for the 10 min postexercise and postrecovery, respectively) and passive recovery conditions (4.1% ± 2.2%, ES = 1.3, 10 min postexercise only).
Although WBV during recovery increased muscle oxygenation, it had little effect in improving subsequent performance compared with a normal active recovery.
Repeated-sprint ability (RSA) is now well accepted as an important fitness component in team-sport performance. It is broadly described as the ability to perform repeated short (~3–4 s, 20–30 m) sprints with only brief (~10–30 s) recovery between bouts. Over the past 25 y a plethora of RSA tests have been trialed and reported in the literature. These range from a single set of ~6–10 short sprints, departing every 20–30 s, to team-sport game simulations involving repeating cycles of walk-jog-stride-sprint movements over 45–90 min. Such a wide range of RSA tests has not assisted the synthesis of research findings in this area, and questions remain regarding the optimal methods of training to best improve RSA. In addition, how RSA test scores relate to player “work rate,” match performance, or both requires further investigation to improve the application of RSA testing and training to elite team-sport athletes.
Jamie Douglas, Daniel J. Plews, Phil J. Handcock and Nancy J. Rehrer
To determine whether a facilitated recovery via cold-water immersion (CWI) after simulated rugby sevens would influence parasympathetic reactivation and repeated-sprint (RS) performance across 6 matches in a 2-d tournament.
Ten male team-sport athletes completed 6 rugby sevens match simulations over 2 d with either postmatch passive recovery (PAS) or CWI in a randomized crossover design. Parasympathetic reactivation was determined via the natural logarithm of the square root of the mean of the sum of the squares of differences between adjacent R-R intervals (ln rMSSD). RS performance was calculated as time taken (s) to complete 6 × 30-m sprints within the first half of each match.
There were large increases in postintervention ln rMSSD between CWI and PAS after all matches (ES 90% CL: +1.13; ±0.21). Average heart rate (HR) during the RS performance task (HRAverage RS) was impaired from baseline from match 3 onward for both conditions. However, HRAverage RS was higher with CWI than with PAS (ES 90% CL: 0.58; ±0.58). Peak HR during the RS performance task (HRPeak RS) was similarly impaired from baseline for match 3 onward during PAS and for match 4 onward with CWI. HRPeak RS was very likely higher with CWI than with PAS (ES 90% CL: +0.80; ±0.56). No effects of match or condition were observed for RS performance, although there were moderate correlations between the changes in HRAverage RS (r 90% CL: –0.33; ±0.14), HRPeak RS (r 90% CL: –0.38; ±0.13), and RS performance.
CWI facilitated cardiac parasympathetic reactivation after a simulated rugby sevens match. The decline in average and peak HR across matches was partially attenuated by CWI. This decline was moderately correlated with a reduction in RS performance.
Ian Craig Perkins, Sarah Anne Vine, Sam David Blacker and Mark Elisabeth Theodorus Willems
We examined the effect of New Zealand blackcurrant (NZBC) extract on high-intensity intermittent running and postrunning lactate responses. Thirteen active males (age: 25 ± 4 yrs, height: 1.82 ± 0.07 m, body mass: 81 ± 14 kg, V̇O2max: 56 ± 4 ml∙kg-1∙min-1, v V̇O2max: 17.6 ± 0.8 km∙h-1) performed a treadmill running protocol to exhaustion, which consisted of stages with 6 × 19 s of sprints with 15 s of low-intensity running between sprints. Interstage rest time was 1 min and stages were repeated with increasing sprint speeds. Subjects consumed capsuled NZBC extract (300 mg∙day-1 CurraNZ; containing 105 mg anthocyanin) or placebo for 7 days (double-blind, randomized, crossover design, wash-out at least 14 days). Blood lactate was collected for 30 min postexhaustion. NZBC increased total running distance by 10.6% (NZBC: 4282 ± 833 m, placebo: 3871 ± 622 m, p = .02), with the distance during sprints increased by 10.8% (p = .02). Heart rate, oxygen uptake, lactate and rating of perceived exertion were not different between conditions for the first 4 stages completed by all subjects. At exhaustion, blood lactate tended to be higher for NZBC (NZBC: 6.01 ± 1.07 mmol∙L-1, placebo: 5.22 ± 1.52 mmol∙L-1, p = .07). There was a trend for larger changes in lactate following 15 min (NZBC: -2.89 ± 0.51 mmol∙L-1, placebo: -2.46 ± 0.39 mmol∙L-1, p = .07) of passive recovery. New Zealand blackcurrant extract (CurraNZ) may enhance performance in sports characterized by high-intensity intermittent exercise as greater distances were covered with repeated sprints, there was higher lactate at exhaustion, and larger changes in lactate during early recovery after repeated sprints to exhaustion.
Conor Taylor, Daniel Higham, Graeme L. Close and James P. Morton
The aim of this study was to test the hypothesis that adding caffeine to postexercise carbohydrate (CHO) feedings improves subsequent high-intensity interval-running capacity compared with CHO alone. In a repeated-measures design, 6 men performed a glycogen-depleting exercise protocol until volitional exhaustion in the morning. Immediately after and at 1, 2, and 3 hr postexercise, participants consumed 1.2 g/kg body mass CHO of a 15% CHO solution, a similar CHO solution but with addition of 8 mg/kg body mass of caffeine (CHO+CAFF), or an equivalent volume of flavored water only (WAT). After the 4-hr recovery period, participants performed the Loughborough Intermittent Shuttle Test (LIST) to volitional exhaustion as a measure of high-intensity interval-running capacity. Average blood glucose values during the 4-hr recovery period were higher in the CHO conditions (p < .005) than in the WAT trial (4.6 ± 0.3 mmol/L), although there was no difference (p = .46) between CHO (6.2 ± 0.8 mmol/L) and CHO+CAFF (6.7 ± 1.0 mmol/L). Exercise capacity during the LIST was significantly longer in the CHO+CAFF trial (48 ± 15 min) than in the CHO (32 ± 15 min, p = .04) and WAT conditions (19 ± 6 min, p = .001). All 6 participants improved performance in CHO+CAFF compared with CHO (95% CI for mean difference = 1–32 min). The study provides novel data by demonstrating that adding caffeine to postexercise CHO feeding improves subsequent high-intensity interval-running capacity, a finding that may be related to higher rates of postexercise muscle glycogen resynthesis previously observed under similar feeding conditions.
David N. Borg, Ian B. Stewart, John O. Osborne, Christopher Drovandi, Joseph T. Costello, Jamie Stanley and Geoffrey M. Minett
circumvent errors in exercise prescription. This is of importance, as errors in prescription that result in an imbalance between training and recovery could lead to nonfunctional overreaching and diminish performance gains. 4 – 6 Traditional heat-based training methods have utilized exercise in a hot
Daniel Viggiani and Jack P. Callaghan
hip extensor musculature in clinical LBP populations arises from the interplay between hip function and LBP. 24 , 25 While LBP can also slow the recovery from muscle fatigue of lumbar spine extensor musculature, 26 – 28 it is not known how LBP affects recovery from hip muscle fatigue. LBP
Fred Brouns, Mikael Fogelholm, Gerrit van Hall, Anton Wagenmakers and Wim H.M. Saris
This study tested the hypothesis that a 3-week oral lactate supplementation affects postexercise blood lactate disappearance in untrained male subjects. Fifteen men were randomly assigned to either a lactate supplementation (n = 8) or a placebo (n = 7) treatment. During the treatment period they drank an oral lactate or a maltodextrin (placebo) supplement twice a day. The lactate drink contained 10 g of lactate as calcium, sodium, and potassium salts. Blood lactate concentrations were studied before, during, and immediately after three exercise tests, both pre-and posttreatment. Peak lactate values for placebo (PL) or lactate (L) treatment groups during different tests were as follows: Test 1 PL, 13.49 ± 3.71; L, 13.70 ± 1.90; Test 2 PL, 12.64 ± 2.32; L, 12.00 ± 2.23; Test 3 PL, 12.29 ± 2.92; L, 11.35 ± 1.38 and were reached 3 min postexercise. The decrease in blood lactate during the long (30- to 45-min) recovery periods amounted to @ 10 mmol/L. Blood lactate changes were highly reproducible. However, a 3-week oral lactate supplementation did not result in differences in lactate disappearance. This study does not support the hypothesis that regular oral lactate intake at rest enhances the removal of lactate during and following exercise, that is, not with the given lactate load and supplementation period.