Search Results

The search for an early biomarker of exercise-induced performance decrement related to muscle damage is an ongoing topic of interest by exercise physiologists, and cell-free DNA (cfDNA) in the plasma is one of the most investigated recently. Small quantities of cfDNA are found both in healthy

myocardial cell injury and necrosis in pathological conditions 8 , 9 but may also stem from increased membrane permeability during exercise. 10 The CK-MB, although considered a cardiac biomarker, is not entirely specific to the myocardium. 10 – 12 The NT-proBNP, on the other hand, is used to monitor heart

standing position, it is difficult to derive thigh and body posture from a hip-worn accelerometer signal. 21 This measurement limitation is important because sedentary behavior dimensions such as sit-to-stand postural transitions and time spent standing may influence metabolic biomarkers and health

addition, or alternative. A variety of biomarkers such as interleukins, creatine kinase, or C-reactive protein have been examined in intermittent sports settings. 1 , 8 Recent studies have shown that some blood parameters were correlated to particular performance traits, such as the increase of creatine

association of PA with SHBG serum levels as one possible biomarker of beneficial effects of PA. The aim of this study was to investigate the association of objectively measured PA with SHBG serum levels in older adults aged 65 years or older under special consideration of potential confounders. Methods Study

acute, vigorous exercise testing at baseline may be contraindicated and in patients who are unfamiliar with exercise. Advances in our assessment and understanding of CVD biomarkers can allow for a safe and effective alternative to exercise testing and serve as an assessment tool to investigate the

adolescent population, also resulting in many injuries and even cardiac emergencies ( 6 , 27 ). Cardiac-specific biomarkers including cardiac troponin I (cTnI) and N-terminal pro B-type natriuretic peptide (NT-proBNP) are presumed to serologically identify cardiac myocyte damage and dysfunction ( 5 ). These

(AWD), on URI incidence and biomarkers of immunity, stress, and nutritional status. We then aimed to explore the relationship between baseline immune, stress and nutritional biomarkers, and URI incidence during HIT in well-trained male cyclists and triathletes. We hypothesized that there would be no

coronary artery disease and myocardial infarction. However, they also reported no relationship of handgrip strength with CVD biomarkers including high-density lipoprotein cholesterol (HDL-C), triglycerides, and fasting glucose ( Xu & Hao, 2017 ), which is inconsistent with findings from previous studies

release dependent on the severity of stretch and stress ( 3 ). The lower detection limits of cTnT and cTnI assays have been greatly reduced in recent years ( 58 ) with new high-sensitivity assays available for both biomarkers. These assays can detect the 99th percentile with a coefficient of variation <10