The effect of oral creatine supplementation on aerobic and anaerobic performance was investigated in 16 elite male rowers during 7-day endurance training. Before and after the daily ingestion of 20 g creatine monohydrate for 5 days (Cr-Group, n = 8) or placebo (Pl-Group, n = 8), subjects performed two exercise tests on a rowing ergometer: (a) incremental exercise consisting of 3-min stage durations and increased by 50 W until volitional exhaustion; (b) an all-out anaerobic exercise performed against a constant load of 7 W/kg. Heart rate and blood lactate concentrations were determined during exercise and recovery. Maximal power output did not significantly differ after the treatment in either group. The mean individual lactate threshold rose significantly after Cr treatment from 314.3 ± 5.0 W to 335.6 ± 7.1 W (p < .01), as compared with 305.0 ± 6.9 W and 308.9 ± 5.9 W (ns), before and after placebo ingestion, respectively. During the anaerobic test, the athletes supplemented with creatine were able to continue rowing longer (mean increase, 12.1 ± 4.5 s; p < .01) than Pl-Group (2.4 ± 8.2 s; ns). No significant differences were found between groups in blood LA after the all-out exercise. The results indicate that in elite rowers, creatine supplementation improves endurance (expressed by the individual lactate threshold) and anaerobic performance, independent of the effect of intensive endurance training.
Alice M. Wallett, Amy L. Woods, Nathan Versey, Laura A. Garvican-Lewis, Marijke Welvaert and Kevin G. Thompson
generally incorporate a period of recovery following intensified training to foster physiological adaptations, with the ultimate aim of leading to an overall improvement in performance. The primary aim of this study was to determine whether a period of intensified endurance training (a mesocycle) would
Mads S. Larsen, Dagmar Clausen, Astrid Ank Jørgensen, Ulla R. Mikkelsen and Mette Hansen
repair, tissue remodeling, and adaptation to endurance training, more so than promoting muscle protein accretion. Previous findings from our laboratory suggest that the ingestion of 0.3 g protein per kg body weight immediately before and after endurance training sessions improves performance and reduces
Daniel L. Blessing, Robert E. Keith, Henry N. Williford, Marjean E. Blessing and Jeff A. Barksdale
The purpose of this study was to determine the effects of an endurance training program on blood lipids and lipoproteins in adolescents. Fifteen males and 10 females, ages 13 to 18 years, underwent pretest evaluations, including physical measurements, nutritional intake, physical working capacity (PWC), and fasting serum lipid and lipoprotein levels. Physical conditioning consisted of a 16-week progressive endurance training (ET) program 40 min·day1 three times per week. Twenty-five males and females matched for age, sex, and race served as controls. Following the conditioning program, the ET group had a significant increase (p < .05) in PWC and a significant decrease (p < .05) in sum of skinfolds and resting heart rate. A significant decrease (p < .05) was also noted for total cholesterol (TC) and the ratio of TC to high density lipoprotein cholesterol (HDL-C) with a significant increase (p < .05) in HDL-C. No differences were found for the control group. The results suggest that 16 weeks of endurance training favorably improves blood lipid profiles in adolescents.
Moritz Schumann, Javier Botella, Laura Karavirta and Keijo Häkkinen
To compare the effects of a standardized endurance-training program with individualized endurance training modified based on the cumulative training load provided by the Polar training-load feature.
After 12 wk of similar training, 24 recreationally endurance-trained men were matched to a training-load-guided (TL, n = 10) or standardized (ST, n = 14) group and continued training for 12 wk. In TL, training sessions were individually chosen daily based on an estimated cumulative training load, whereas in ST the training was standardized with 4–6 sessions/wk. Endurance performance (shortest 1000-m running time during an incremental field test of 6 × 1000 m) and heart-rate variability (HRV) were measured every 4 wk, and maximal oxygen consumption (VO2max) was measured during an incremental treadmill test every 12 wk.
During weeks 1–12, similar changes in VO2max and 1000-m time were observed in TL (+7% ± 4%, P = .004 and –6% ± 4%, P = .069) and ST (+5% ± 7%, P = .019 and –8% ± 5%, P < .001). During wk 13–24, VO2max statistically increased in ST only (3% ± 4%, P = .034). The 1000-m time decreased in TL during wk 13–24 (–9% ± 5%, P = .011), but in ST only during wk 13–20 (–3% ± 2%, P = .003). The overall changes in VO2max and 1000-m time during wk 0–24 were similar in TL (+7% ± 4%, P = .001 and –9% ± 5%, P = .011) and ST (+10% ± 7%, P < .001 and –13% ± 5%, P < .001). No between-groups differences in total training volume and frequency were observed. HRV remained statistically unaltered in both groups.
The main finding was that training performed according to the cumulative training load led to improvements in endurance performance similar to those with standardized endurance training in recreational endurance runners.
Daniel J. Davies, Kenneth S. Graham and Chin Moi Chow
The use of daytime napping as a recovery tool following exercise is virtually unexplored. The objective of this study was to assess the quality of daytime nap sleep following endurance training in an athletic population, and to appraise the optimal circadian timing of the nap and the time interval between training and the nap.
Six physically trained male subjects (22.5 ± 2.4 y) performed four separate standardized 90-min endurance training sessions followed by a 90-min daytime nap either 1 or 2 h after training (time interval), commencing at either 10:30 or 11:30 (circadian timing). During the nap, sleep was monitored using polysomnography. Subjective measurements of sleep quality, alertness and preparedness to train following a nap were recorded using a visual analog scale.
The duration of slow wave sleep (SWS) was significantly greater during the 11:30 naps (13.7 ± 9.0 min) compared with the 10:30 naps (6.9 ± 8.8 min) (P = .049). There was no significant difference in SWS duration between a 1-h (10.6 ± 10.2 min) or 2-h (10.0 ± 9.0 min) time interval between training and the nap (P = .82). No other sleep variables differed significantly according to circadian timing or time interval.
Recovery naps commenced later in the morning contain more SWS than earlier naps. The data imply that daytime naps have a potential role as a valuable recovery tool following endurance exercise, given the suggested energy restorative functions of SWS.
Rudolph H. Dressendorfer, Stewart R. Petersen, Shona E. Moss Lovshin and Carl L. Keen
This study examined the effects of intense endurance training on basal plasma and 24-hour urinary calcium (Ca), magnesium (Mg), iron (Fe), zinc (Zn), and copper (Cu) levels in 9 male competitive cyclists. The supervised training program followed a baseline period and included a volume phase (6 weeks, averaging 87% of maximal heart rate [HRmax]), an interval phase (18 days, 100% of HRmax), and a 10-day unloading taper. The primary training outcome measure was 20-km time-trial cycling performance. Subjects ate unrestricted diets and maintained their weight. Compared to baseline, performance improved significantly (p < .05), while mineral metabolism was not significantly different after the volume phase. However, after the interval phase, renal Ca excretion increased (p < .05) and plasma Ca fell slightly below the clinical norm. As compared to the interval phase, urinary Ca decreased (p < .05), plasma Ca increased (p < .05), and performance further improved (p < .05) after the taper. Whereas Mg, Fe, Zn, and Cu metabolism remained unchanged throughout the study, greater renal Ca excretion was associated with very high intensity interval training.
Theofanis Tzatzakis, Konstantinos Papanikolaou, Dimitrios Draganidis, Panagiotis Tsimeas, Savvas Kritikos, Athanasios Poulios, Vasiliki C. Laschou, Chariklia K. Deli, Athanasios Chatzinikolaou, Alexios Batrakoulis, Georgios Basdekis, Magni Mohr, Peter Krustrup, Athanasios Z. Jamurtas and Ioannis G. Fatouros
training is extensively utilized to improve players’ ability to perform repeated high-intensity actions and delay fatigue. 6 Speed-endurance training emphasizes the activation of nonoxidative energy pathways using repeated sprints consisting of multiple changes of direction, a protocol also known in
Thomas W. Jones, Ian H. Walshe, David L. Hamilton, Glyn Howatson, Mark Russell, Oliver J. Price, Alan St Clair Gibson and Duncan N. French
To compare anabolic signaling responses to differing sequences of concurrent strength and endurance training in a fed state.
Eighteen resistance-trained men were randomly assigned to the following experimental conditions: strength training (ST), strength followed by endurance training (ST-END), or endurance followed by strength training (END-ST). Muscle tissue samples were taken from the vastus lateralis before each exercise protocol, on cessation of exercise, and 1 h after cessation of strength training. Tissue was analyzed for total and phosphorylated (p-) signaling proteins linked to the mTOR and AMPK networks.
Strength-training performance was similar between ST, ST-END, and END-ST. p-S6k1 was elevated from baseline 1 h posttraining in ST and ST-END (both P < .05). p-4E-BP1 was significantly lower than baseline post-ST (P = .01), whereas at 1 h postexercise in the ST-END condition p-4E-BP1 was significantly greater than postexercise (P = .04). p-ACC was elevated from baseline both postexercise and 1 h postexercise (both P < .05) in the END-ST condition. AMPK, mTOR, p38, PKB, and eEF2 responded similarly to ST, ST-END, and END-ST. Signaling responses to ST, ST-END, and END were largely similar. As such it cannot be ascertained which sequence of concurrent strength and endurance training is most favorable in promoting anabolic signaling.
In the case of the current study an acute bout of concurrent training of differing sequences elicited similar responses of the AMPK and mTOR networks.
Kevin A. Jacobs, David R. Paul, Ray J. Geor, Kenneth W. Hinchcliff and W. Michael Sherman
The purpose of the current study was to examine the influence of dietary composition on short-term endurance training–induced adaptations of substrate partitioning and time trial exercise performance. Eight untrained males cycled for 90 min at ~54% aerobic capacity while being infused with [6,62H]glucose before and after two 10-d experimental phases separated by a 2-week washout period. Time trial performance was measured after the 90-min exercise trials before and after the 2nd experimental phase. During the first 10-d phase, subjects were randomly assigned to consume either a high carbohydrate or high fat diet while remaining inactive (CHO or FAT). During the second 10-d phase, subjects consumed the opposite diet, and both groups performed identical daily supervised endurance training (CHO+T or FAT+T). CHO and CHO+T did not affect exercise metabolism. FAT reduced glucose flux at the end of exercise, while FAT+T substantially increased whole body lipid oxidation during exercise and reduced glucose flux at the end of exercise. Despite these differences in adaptation of substrate use, training resulted in similar improvements in time trial performance for both groups. We conclude that (a) 10-d high fat diets result in substantial increases in whole body lipid oxidation during exercise when combined with daily aerobic training, and (b) diet does not affect short-term training-induced improvements in high-intensity time trial performance.