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Naroa Etxebarria, Nicole A. Beard, Maree Gleeson, Alice Wallett, Warren A. McDonald, Kate L. Pumpa, and David B. Pyne

for biomarkers, which represent GI permeability, included lipopolysaccharide (LPS), lipopolysaccharide-binding protein, and intestinal fatty-acid-binding protein (i-FABP). All biomarkers were analyzed in duplicate, as previously described ( Wallett et al., 2020 ). Statistical Analysis The descriptive

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Alice Wallett, Andrew McKune, David Pyne, David Bishop, Olivier Girard, Philo Saunders, and Julien Périard

associated with maximum rate of oxygen consumption, plasma intestinal fatty acid binding protein concentration ([I-FABP]), and GI permeability (as measured by the ratio of L-lactulose/rhamnose) increased by 72% and 59%, respectively, relative to rest ( Pugh et al., 2017 ). Similarly, running at 80% of best

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Alice M. Wallett, Naroa Etxebarria, Nicole A. Beard, Philo U. Saunders, Marijke Welvaert, Julien D. Périard, Andrew J. McKune, and David B. Pyne

), an acute phase response protein that mediates the rise in LPS, and intestinal fatty acid–binding protein (I-FABP), an early GI-ischemia marker. 10 While there is evidence supporting the premise that heat stress mediates exercise-induced endotoxemia, specific GI responses to exercise in the heat at

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Ben J. Lee, Tessa R. Flood, Ania M. Hiles, Ella F. Walker, Lucy E.V. Wheeler, Kimberly M. Ashdown, Mark E.T. Willems, Rianne Costello, Luke D. Greisler, Phebe A. Romano, Garrett W. Hill, and Matthew R. Kuennen

(210 mg anthocyanins per day) on small intestinal permeability (dual sugar absorption test), plasma intestinal epithelial injury (serum intestinal fatty acid-binding protein [I-FABP]), microbial translocation (serum lipopolysaccharide-binding protein [LBP]), and soluble CD14 [sCD14]) in conjunction

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Ricardo J.S. Costa, Vera Camões-Costa, Rhiannon M.J. Snipe, David Dixon, Isabella Russo, and Zoya Huschtscha

fatty acid binding protein (I-FABP) concentration ( Costa et al., 2019b ; Costa et al., 2017b ). In addition, exercise stress has the potential to reduce gastrointestinal transit and enterocyte cell activity through mesenteric and submucosal plexus deactivation linked with the natural stress response

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SILVA. Blood samples were collected pre- and post-exercise to determine I-FABP, cortisol, and cytokine profiles. Markers of physiological and thermoregulatory strain, and gastrointestinal symptoms (GIS) were measured throughout. Correlations and β-diversity analysis between amplicon sequence variants

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Ricardo J.S. Costa, Pascale Young, Samantha K. Gill, Rhiannon M.J. Snipe, Stephanie Gaskell, Isabella Russo, and Louise M. Burke

standardize ( Pugh et al., 2019 ; Roberts et al., 2016 ; Zadow et al., 2021 ). Figure 2 —Exercise-associated disturbances to markers of gastrointestinal integrity, systemic responses, and GIS. Plasma (a) I-FABP, (b) sCD14, (c) LBP, (d) gram-negative bacterial endotoxin, (e) antiendotoxin IgM, (f

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Isabella Russo, Paul A. Della Gatta, Andrew Garnham, Judi Porter, Louise M. Burke, and Ricardo J.S. Costa

, intestinal fatty-acid-binding protein [I-FABP] and sCD14 enzyme-linked immunosorbent assay [ELISA], and multiplex system for systemic inflammatory profile) as previously reported. 6 , 15 , 22 The CVs for ELISAs were ≤6.1%, and for cytokine, profile multiplex was 16.0%. Breath samples (20 mL) were analyzed

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samples were collected pre- and post-exercise, and during recovery to determine plasma I-FABP and cortisol. Breath samples were collected every 30 min in recovery for 3 h to determine H 2 (malabsorption marker) responses. DEH resulted in higher heart rate (p = .038; 158 vs 152 bpm), RPE (p = .001; 15 vs

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Kirsty M. Reynolds, Tom Clifford, Stephen A. Mears, and Lewis J. James

Gut rumbling Flatulence Abdominal discomfort Potato and water consumption led to greater overall GI symptoms, abdominal bloating, abdominal pain, and abdominal discomfort at 120 min ( p  < .05) compared with gel and water There was no correlation between plasma I-FABP concentrations and GI symptoms