Prescribing training loads for endurance athletes often incorporates the measurement of the blood lactate response to incremental exercise in conjunction with heart rate (HR), oxygen consumption ( V ˙ O 2 ), and exercise intensity, and the subsequent calculation of blood lactate thresholds
Pitre C. Bourdon, Sarah M. Woolford, and Jonathan D. Buckley
Pedro L. Valenzuela, Javier S. Morales, Carl Foster, Alejandro Lucia, and Pedro de la Villa
The lactate threshold (LT), usually defined as the maximum workload that precedes an exponential rise in blood lactate values during an incremental test, is one of the most popular markers of the so-called anaerobic transition. This marker has been extensively used as a predictor of endurance
Wolf-Stephan Rudi, Florian Maier, Dominik Schüttler, Antonia Kellnar, Anna Katharina Strüven, Wolfgang Hamm, and Stefan Brunner
, respectively, P = .005, NM vs FFP2 P = .007) (Figure 1C ). Figure 1 —The iLT, RPE, and peak performance in different conditions: (A) power (in Watts) at individual lactate threshold calculated by the Dickhuth method and at 4 mM of lactate concentration according to Mader in different conditions, NM versus
Fiona Iredale, Frank Bell, and Myra Nimmo
Fourteen sedentary 50- to 55-year-old men were exercised to exhaustion using an incremental treadmill protocol. Mean (±SEM) peak oxygen uptake (V̇O2peak) was 40.5 ± 1.19 ml · kg1 · min−1, and maximum heart rate was 161 ± 4 beats · min−1. Blood lactate concentration was measured regularly to identify the lactate threshold (oxygen consumption at which blood lactate concentration begins to systematically increase). Threshold occurred at 84 ± 2% of V̇O2peak. The absolute lactate value at threshold was 2.9 ± 0.2 mmol · L−1. On a separate occasion, 6 subjects exercised continuously just below their individual lactate thresholds for 25 min without significantly raising their blood lactate levels from the 10th minute to the 25th. The absolute blood lactate level over the last 20 min of the steady-state test averaged 3.7 ± 1.2 mmol · L−1. This value is higher than that elicited at the threshold in the incremental test because of the differing nature of the protocols. It was concluded that although the lactate threshold occurs at a high percentage of V̇O2peak, subjects are still able to sustain exercise at that intensity for 25 min.
Peter Pfitzinger and Patty Freedson
Part 2 reviews the literature concerning the lactate threshold in children. An analysis is presented comparing children to adults regarding responses to submaximal exercise, and the lactate threshold as a percentage of VO2max. Possible explanations for lower blood lactate concentrations during submaximal exercise in children are considered.
Sijie Tan, Chunhua Yang, and Jianxiong Wang
The purpose of this study was to apply the lactate threshold concept to develop a more evidence-informed exercise program for obese children. 60 obese children (26 girls and 34 boys, age: 9–10 years, BMI: 25.4 ± 2.2kg/m2) were recruited and half of them were randomly selected to be trained for eight weeks with a controlled exercise intensity at lactate threshold. The trained children achieved significant improvements on their body composition and functional capacity compared with the control group. The findings suggested that the training program with intensity at lactate threshold is effective and safe for 9–10 year old children with obesity.
Samuel Chalmers, Adrian Esterman, Roger Eston, and Kevin Norton
The purpose of this study was to test the reliability and validity of 2 standardized methods for calculating speed at the second lactate-threshold point (LT2) based on the preexisting Dmax (LTD) and modified Dmax (LTMOD) procedures.
13 trained male road runners and triathletes completed 2 incremental laboratory running tests to determine LT2, followed by separate time trials (5, 10, 15 km) on an outdoor running track. Two new methods were proposed for calculating the speed at LT2: (1) the single standardized lactate threshold (LTSDs) and (2) the paired standardized lactate threshold (LTSDp) for quantifying changes over time.
The LTSDs and LTSDp methods had high relative (ICC ≥ .98) and absolute (CV ≤ 1.9%) reliability in identifying the speed at LT2. The speed at LT2 according to the LTSDs and LTSDp methods had a strong correlation and was not different to the performance speed during the 10- and 15-km time trials (≤2.3%; ρc > 0.8; P > .05). The following natural logbased formula was created to estimate the percentage of LT2 speed (using the LTSDs method) that could be sustained for events ~15–75 min: y = –7.256(ln x) + 157.64, where y = %LT2 speed, x = time-trial performance (s), and ln = natural log.
The standardized methods are reliable for determining LT2. The LTSDs and LTSDp methods for calculating the speed at LT2 from a near-maximal incremental test calculated speeds similar to those exhibited in 10- and 15-km running time trials. A prediction equation for estimating the percentage of LT2 that can be sustained for events of ~15–75 min was generated.
K. Fiona Iredale and Myra A. Nimmo
Thirty-three men (age 26–55 years) who did not exercise regularly were exercised to exhaustion using an incremental treadmill protocol. Blood lactate concentration was measured to identify lactate threshold (LT, oxygen consumption at which blood lactate concentration begins to systematically increase). The correlation coefficient for LT (ml · kg−1 · min−1) with age was not significant, but when LT was expressed as a percentage of peak oxygen consumption (VO2 peak), the correlation was r = +.69 (p < .01). This was despite a lack of significant correlation between age and VO2 peak (r = −.33). The correlation between reserve capacity (the difference between VO2 peak and LT) and age was r = −.73 (p < .01 ), and reserve capacity decreased at a rate of 3.1 ml · kg−1 · min−1 per decade. It was concluded that the percentage of VO2 peak at which LT occurs increases progressively with age, with a resultant decrease in reserve capacity.
Jacky J. Forsyth, Chris Mann, and James Felix
In rowing ergometry, blood for determining lactate concentration can be removed from the toe tip without the rower having to stop. The purpose of the study was to examine whether sampling blood from the toe versus the earlobe would affect lactate threshold (Tlac) determination.
Ten physically active males (mean ± age 21.2 ± 2.3 y; stature 179.2 ± 7.5 cm; body mass 81.7 ± 12.7 kg) completed a multistage, 3 min incremental protocol on the Concept II rowing ergometer. Blood was sampled simultaneously from the toe tip and earlobe between stages. Three different methods were used to determine Tlac.
There were wider variations due to the method of Tlac determination than due to the sample site; for example, ANOVA results for power output were F(1.25, 11.25) = 11.385, P = .004 for method and F(1, 9) = 0.633, P = .45 for site. The greatest differences in Tlac due to sample site in rowing occurred when Tlac was determined using an increase in blood lactate concentration by >1 mmol/L from baseline (TlacΔ1).
The toe tip can be used as a suitable sample site for blood collection during rowing ergometry, but caution is needed when using the earlobe and toe tip interchangeably to prescribe training intensities based on Tlac, especially when using TlacΔ1 or at lower concentrations of lactate.
Samuel Chalmers, Adrian Esterman, Roger Eston, and Kevin Norton
Athletes often seek the minimum required time that might elicit a physiological or performance change. It is reasonable to suggest that heat training may improve aerobic-based performance in mild conditions. Therefore, rather than providing a traditional heat-exposure stimulus (ie, 7–10 × 60–100 min sessions), the current article details 2 studies that aimed to determine the effect of brief (≤240 min exposure) heat training on the second lactate threshold (LT2) in mild conditions.
Forty-one participants completed 5 (study 1, n = 18) or 4 (study 2, n = 23) perceptually regulated treadmill exercise training sessions in 35°C and 30% relative humidity (RH) (experimental group) or 19°C and 30% RH (control group). Preincremental and postincremental exercise testing occurred in mild conditions (19°C and 30% RH). Linear mixed-effects models analyzed the change in LT2.
Heat training did not substantially change LT2 in either study 1 (+1.2%, d = 0.38, P = .248) or study 2 (+1.9%, d = 0.42, P = .163). LT2 was not substantially changed in the control group in study 1 (+1.3%, d = 0.43, P = .193), but a within-group change was evident in study 2 (+2.3%, d = 1.04, P = .001).
Brief heat training was inadequate to improve the speed at LT2 in mild conditions more than comparable training in mild conditions. The brief nature of the heattraining protocol did not allow adaptations to develop to the extent required to potentially confer a performance advantage in an environment that is less thermally stressful than the training conditions.