The purpose of this study was to assess the validity and reliability of the MedGem™ device to measure resting metabolic rate (RMR) in children. Subjects included 59 children (29 boys, 30 girls; mean age, 11.0 ± 0.2 y). Subjects were given 4 RMR tests during 1 test session, cconsisting of 2 Douglas bag and 2 MedGem tests, in random counterbalanced order. No significant differences were found between Douglas bag and MedGem systems for oxygen consumption (209 ± 5 and 213 ± 5 mL/min, respectively, P = 0.106, r = 0.911, mean ± standard deviation absolute difference 3.72 ± 17.40 mL/min) or RMR (1460 ± 39 and 1477 ± 35 kcal/d, P = 0.286, r = 0.909, mean ± standard deviation absolute difference 17.4 ± 124 kcal/d). Standard error of estimates for oxygen consumption and RMR were 17.4 mL/min and 124 kcal/d, respectively. In conclusion, these data indicate that the MedGem is a reliable and valid system for measuring oxygen consumption and RMR in children.
Search Results
Validation of a New Handheld Device for Measuring Resting Metabolic Rate and Oxygen Consumption in Children
David C. Nieman, Melanie D. Austin, Shannon M. Chilcote, and Laura Benezra
Exercising Fasting or Fed to Enhance Fat Loss? Influence of Food Intake on Respiratory Ratio and Excess Postexercise Oxygen Consumption After a Bout of Endurance Training
Antonio Paoli, Giuseppe Marcolin, Fabio Zonin, Marco Neri, Andrea Sivieri, and Quirico F. Pacelli
Exercise and nutrition are often used in combination to lose body fat and reduce weight. In this respect, exercise programs are as important as correct nutrition. Several issues are still controversial in this field, and among them there are contrasting reports on whether training in a fasting condition can enhance weight loss by stimulating lipolytic activity. The authors’ purpose was to verify differences in fat metabolism during training in fasting or feeding conditions. They compared the effect on oxygen consumption (VO2) and substrate utilization, estimated by the respiratory-exchange ratio (RER), in 8 healthy young men who performed the same moderate-intensity training session (36 min of cardiovascular training on treadmill at 65% maximum heart rate) in the morning in 2 tests in random sequence: FST test (fasting condition) without any food intake or FED test (feeding condition) after breakfast. In both cases, the same total amount and quality of food was assumed in the 24 hr after the training session. The breakfast, per se, increased both VO2 and RER significantly (4.21 vs. 3.74 and 0.96 vs. 0.84, respectively). Twelve hours after the training session, VO2 was still higher in the FED test, whereas RER was significantly lower in the FED test, indicating greater lipid utilization. The difference was still significant 24 hr after exercise. The authors conclude that when moderate endurance exercise is done to lose body fat, fasting before exercise does not enhance lipid utilization; rather, physical activity after a light meal is advisable.
Sex-Related Differences in Oxygen Consumption Recovery After High-Intensity Rowing Exercise During Childhood and Adolescence
Joffrey Bardin, Hugo Maciejewski, Allison Diry, Claire Thomas, and Sébastien Ratel
, although some comparative pediatric data do exist ( 18 ). From a cardiorespiratory perspective, some studies have reported a faster recovery of oxygen consumption ( V ˙ O 2 ) in prepubertal boys compared with men after high-intensity exercise ( 8 , 15 , 21 , 40 ). For instance, using biexponential modeling
Temporal Location of High-Intensity Interval Training in Cycling Does Not Impact the Time Spent Near Maximal Oxygen Consumption
James R. Mckee, Bradley A. Wall, and Jeremiah J. Peiffer
. However, for aerobic adaptation (ie, increases in cardiac output and maximal oxygen consumption [ V ˙ O 2 max ]), the total time an individual spends at or near their V ˙ O 2 max is an important consideration. 5 , 6 Many studies have examined the influence of interval structure on the time spent at or
Five-Minute Power-Based Test to Predict Maximal Oxygen Consumption in Road Cycling
Sebastian Sitko, Rafel Cirer-Sastre, Francisco Corbi, and Isaac López-Laval
In recent years, multiple performance factors have been identified in road cycling. 1 Among the physiological determinants of performance, maximal oxygen consumption (VO 2 max) adjusted to body mass can be highlighted as a key parameter of cardiorespiratory fitness. 2 Normally, gas exchange
Is the Polar M430 a Valid Tool for Estimating Maximal Oxygen Consumption in Adult Females?
Kevin E. Miller, Timothy R. Kempf, Brian C. Rider, and Scott A. Conger
Maximal oxygen consumption ( V ˙ O 2 max ) plays an integral role in health and wellness. The assessment of V ˙ O 2 max can be used to prescribe exercise intensity, evaluate the progress of an exercise program, and evaluate endurance performance potential ( American College of Sports Medicine
The Acute Physiological and Perceptual Effects of Individualizing the Recovery Interval Duration Based Upon the Resolution of Muscle Oxygen Consumption During Cycling Exercise
Christopher R.J. Fennell and James G. Hopker
reflects the ratio of oxygen (O 2 ) delivery to the working muscle and muscle oxygen uptake in the capillary beds. 17 The recovery of muscle oxygen consumption ( m V ˙ O 2 ) considers the condition of the exercising muscle, as measurements are derived directly from the muscle body. It has been suggested
Comparison of a Traditional Graded Exercise Protocol With a Self-Paced 1-km Test to Assess Maximal Oxygen Consumption
Roland van den Tillaar, Erna von Heimburg, and Guro Strøm Solli
Maximal oxygen consumption (VO 2 max) is defined as the highest rate at which oxygen can be taken up and utilized by the body during intensive exercise. 1 The VO 2 max test is frequently used as a measure of the cardiorespiratory fitness level of an individual or as a physiological marker for
Four Weeks of Intensified Training Enhances On-Ice Intermittent Exercise Performance and Increases Maximal Oxygen Consumption of Youth National-Team Ice Hockey Players
Jan Sommer Jeppesen, Jeppe F. Vigh-Larsen, Mikkel S. Oxfeldt, Niklas M. Laustsen, Magni Mohr, Jens Bangsbo, and Morten Hostrup
-intensity intermittent exercise performance, sprint, and agility performance conducted on-ice as well as maximum oxygen consumption ( V ˙ O 2 max ), muscle strength, rate of force development, and body composition in youth national team ice hockey players. We hypothesized that 4 weeks of SET would enhance high
Excess Postexercise Oxygen Consumption After Aerobic Exercise Training
Darlene A. Sedlock, Man-Gyoon Lee, Michael G. Flynn, Kyung-Shin Park, and Gary H. Kamimori
Literature examining the effects of aerobic exercise training on excess postexercise oxygen consumption (EPOC) is sparse. In this study, 9 male participants (19–32 yr) trained (EX) for 12 wk, and 10 in a control group (CON) maintained normal activity. VO2max, rectal temperature (Tre), epinephrine, norepinephrine, free fatty acids (FFA), insulin, glucose, blood lactate (BLA), and EPOC were measured before (PRE) and after (POST) the intervention. EPOC at PRE was measured for 120 min after 30 min of treadmill running at 70% VO2max. EX completed 2 EPOC trials at POST, i.e., at the same absolute (ABS) and relative (REL) intensity; 1 EPOC test for CON served as both the ABS and REL trial because no significant change in VO2max was noted. During the ABS trial, total EPOC decreased significantly (p < .01) from PRE (39.4 ± 3.6 kcal) to POST (31.7 ± 2.2 kcal). Tre, epinephrine, insulin, glucose, and BLA at end-exercise or during recovery were significantly lower and FFA significantly higher after training. Training did not significantly affect EPOC during the REL trial; however, epinephrine was significantly lower, and norepinephrine and FFA, significantly higher, at endexercise after training. Results indicate that EPOC varies as a function of relative rather than absolute metabolic stress and that training improves the efficiency of metabolic regulation during recovery from exercise. Mechanisms for the decreased magnitude of EPOC in the ABS trial include decreases in BLA, Tre, and perhaps epinephrine-mediated hepatic glucose production and insulin-mediated glucose uptake.
