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Scott Cheatham, Morey J. Kolber and Michael P. Ernst


Pulse oximetry has become mobile with the use of smartphone and Bluetooth wireless technology. This technology offers many benefits but has not been extensively studied. There is a need to further validate its clinimetric properties for health professionals to provide proper guidance to patients.


This investigation assessed the concurrent validity of the iSpO2 pulse oximeter against a traditional pulse oximeter in measuring short-term resting blood oxygen saturation (SpO2) and pulse rate.


Observational study of reliability.


University kinesiology laboratory.


Thirty healthy, recre-ationally active adults (18 men, 12 women; mean age = 25.7 ± 5.46 years, mean height = 170.3cm ± 9.51, mean body mass = 76.4 kg ± 19.33).


Resting measurement of SpO2 and pulse rate using the iSpO2 pulse oximeter with the iPad Mini and a traditional pulse oximeter with Bluetooth.

Main Outcome Measure:

Resting SpO2 and pulse rate were concurrently measured over 5 min.


The concurrent validity between the iSpO2 and traditional pulse oximeter was moderate for measuring SpO2, intraclass correlation coeffcient (ICC)(3, 1) = .73, SEM = 0.70%, and good for pulse rate, ICC(3, 1) = .97, SEM = 1.74 beats per minute (bpm). The minimal detectable change at the 95% confidence interval for both instruments suggests that there may be 1.94% disagreement for SpO2 and 4.82 bpm disagreement between pulse oximetry methods. The 95% limits of agreement (LoA) for measuring SpO2 suggests that the iSpO2 and traditional pulse oximeters may vary -0.28 ± 1.98%, or approximately 2%. The 95% LoA for measuring pulse rate suggests that the iSpO2 and traditional pulse oximeter may vary 1.74 ± 4.98 bpm, potentially upward of 6 bpm. On the basis of the results of the LoA, it appears that there may be a slight systematic bias between the two devices, with the traditional pulse oximeter producing higher pulse rates than the iSpO2.


The findings suggest that both instruments may be beneficial for indirect short-term measurements of resting SpO2 and pulse rate.

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Laurent Trincat, Xavier Woorons and Grégoire P. Millet


Repeated-sprint training in hypoxia (RSH) has been shown as an efficient method for improving repeated-sprint ability (RSA) in team-sport players but has not been investigated in swimming. We assessed whether RSH with arterial desaturation induced by voluntary hypoventilation at low lung volume (VHL) could improve RSA to a greater extent than the same training performed under normal breathing (NB) conditions.


Sixteen competitive swimmers completed 6 sessions of repeated sprints (2 sets of 16 × 15 m with 30 s send-off) either with VHL (RSH-VHL, n = 8) or with NB (RSN, n = 8). Before and after training, performance was evaluated through an RSA test (25-m all-out sprints with 35 s send-off) until exhaustion.


From before to after training, the number of sprints was significantly increased in RSH-VHL (7.1 ± 2.1 vs 9.6 ± 2.5; P < .01) but not in RSN (8.0 ± 3.1 vs 8.7 ± 3.7; P = .38). Maximal blood lactate concentration ([La]max) was higher after than before in RSH-VHL (11.5 ± 3.9 vs 7.9 ± 3.7 mmol/L; P = .04) but was unchanged in RSN (10.2 ± 2.0 vs 9.0 ± 3.5 mmol/L; P = .34). There was a strong correlation between the increases in the number of sprints and in [La]max in RSH-VHL only (R = .93, P < .01).


RSH-VHL improved RSA in swimming, probably through enhanced anaerobic glycolysis. This innovative method allows inducing benefits normally associated with hypoxia during swim training in normoxia.

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Ben J. Lee and Charles Douglas Thake

saturation (SpO 2 ) and disrupting homeostasis. 11 Anecdotal reports indicate that this method is now used with a view to increasing the aerobic training stimulus when exercising with BWS in rehabilitation settings (eg, with professional football players). The rationale for this approach is that due to the

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Brian Killinger, Jakob D. Lauver, Luke Donovan and John Goetschius

rehabilitation is to establish whether these acute responses can be induced within the lower-leg muscles of CAI patients. Therefore, the primary purposes of this study were to examine the effects of BFR on muscle activation and oxygen saturation during submaximal eversion and dorsiflexion resistance exercises in

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Pedro L. Valenzuela, Guillermo Sánchez-Martínez, Elaia Torrontegi, Javier Vázquez-Carrión, Manuela González, Zigor Montalvo and Grégoire P. Millet

. Perceptual Response The overall rating of perceived exertion (overall RPE) and the fatigue perceived locally on the legs (legs RPE) were determined using Borg CR-10 scale at the end of the training session. Oxygen Kinetics Pulse oxygen saturation (SpO 2 ) was assessed with a finger oximeter (Dimed, Vizcaya

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Philippe Richard and François Billaut

calculated using the tissue saturation index (TSI [%]) as [O 2 HbMb] divided by ([O 2 HbMb] + [HHbMb]) × 100. The [HHbMb] signal was also taken as an indicator of tissue deoxygenation because this variable is less sensitive than [O 2 HbMb] to perfusion variations and abrupt blood volume changes during

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Jessica Barrett, Alicia Pike and Stephanie Mazerolle

athletics websites of NCAA Division I universities (listed on the NCAA website). We recruited participants until data saturation was met, which occurred at 15 (including 1 from the pilot study). Participants were, on average, 33 years old (±9, range = 23–58) with an average of 11 years of experience (±9

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David Morawetz, Tobias Dünnwald, Martin Faulhaber, Hannes Gatterer and Wolfgang Schobersberger

explicitly prohibited the application of supplemental oxygen, but it is prohibited by the FIS and the International Olympic Committee (IOC) for their official competitions. 14 – 16 Hyperoxia improves arterial partial pressure of oxygen (PaO 2 ), oxygen saturation of hemoglobin (SaO 2 ), and the amount of O 2

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Daniel G. Hursh, Marissa N. Baranauskas, Chad C. Wiggins, Shane Bielko, Timothy D. Mickleborough and Robert F. Chapman

Endurance exercise performance in hypoxia is impaired partly due to declines in arterial oxyhemoglobin saturation (S p O 2 ), 1 which may limit the delivery of oxygen to exercising muscle. A well-established mechanism of defending S p O 2 during hypoxic exercise is to increase minute ventilation

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Myriam Paquette, François Bieuzen and François Billaut

than half the distance between the emitter and the detector (25 mm). The raw muscle O 2 saturation (SmO 2 ) and total hemoglobin concentration ([THb]) signals were treated using a smooth spline filter to reduce the noise created by movement. During exercise, SmO 2 represents the balance between O 2